Match each clinical feature of constrictive pericarditis with its underlying pathophysiological mechanism: Clinical Features: 1. Jugular venous distension 2. Pericardial knock 3. Ascites 4. Exercise intolerance Mechanisms: - Impaired right ventricular filling leading to elevated systemic venous pressure - Abrupt cessation of early diastolic ventricular filling by a rigid pericardium - Chronic systemic venous congestion leading to peritoneal fluid accumulation - Inability to increase stroke volume and cardiac output during exertion

These pairings connect the clinical features of constriction with the mechanisms described in the lecture. Raised systemic venous pressure manifests as jugular venous distension; chronic congestion can produce ascites. A pericardial knock is linked to sudden cessation of rapid ventricular filling in early diastole, and exercise intolerance reflects limited cardiac output. Recognizing these relationships helps distinguish signs of venous congestion from those of low output. **Key takeaway:** The signs of constrictive pericarditis arise from restricted filling, venous congestion, and reduced output. **Glossary:** **Systemic venous pressure**: pressure in the veins returning blood from the body. **Cardiac output**: blood pumped by the heart over time.